Leptomycin B reduces primary and acquired resistance of gefitinib in lung cancer cells

Description:

Technology Summary

The use of EGFR inhibitors in the treatment of NSCLC is well established, even being used as a first-line treatment for certain drugs like Gefitinib and Osimertinib. However, eventual drug-resistance by the tumor to these treatments remains a problem. This technology showcases how CRM1 inhibitor Leptomycin B can potentiate the EGFR inhibitor Gefitinib, diminish resistance, and even resensitize tumor cells to Gefitinib. The technology is a method for using CRM1 inhibitors alongside EGFR inhibitors in lung-cancer treatment to mitigate acquired drug resistance, resensitize resistant tumors, and enhance the potency of the EGFR inhibitor.

 

Reference Number

D-1448

Market Applications

•      Therapeutics

•      Oncology

Features, Benefits & Advantages

•       The toxic effects of leptomycin B are less severe in a combinational therapy

•       Reversal of Gefitinib resistance in NSCLC

•       Prevents Gefitinib resistance in NSCLC

 

Intellectual Property

A provisional patent with a claim set has been filed recently, 4/3/18.

 

Development Stage

The disclosed technology has been tested in-vitro in cancer cell lines.

 

Researchers

Dr. Weimin Gao, Associate Professor, Department of Environmental Toxicology and the Institute of Environmental and Human Health, Texas Tech University System.

 

Keywords

Gefitinib, leptomycin B, cancer, NSCLC, treatment, combinational

 

Patent Information:
Category(s):
Cancer Therapeutics
For Information, Contact:
Hutton Jones
Texas Tech Office of Research Commercialization
806-742-4105
william.h.jones@ttu.edu
Inventors:
Weimin Gao
Zhongwei Liu
Keywords: